i keep the two on separate days so if something goes wrong i have a chance of knowing which
#cagrilintide 2025-04-19
i said earlier this year that monotherapy did nothing for me, and at a higher dose it did something
amylin is about slowed gastric emptying and satiety signalling, thats the short version and im no pharmacologist
has REDEFINE reported or are people quoting earlier work
the fullness effect hasnt faded over 4 months, unlike my semaglutide appetite curve, thats one data point
15 in 5ml gives 2mg/ml which is what i use, purely because the units come out round
back after 9 months, is monotherapy still a minority thing here
combination GI was worse than either alone for me, which is not what i had read anywhere
anyone holding at 10 for a while before stepping
while im here the reason nausea reads differently is timing, mine arrived on the second day rather than the first
if youre used to GLP-1 titration steps this feels absurdly cautious and i still do it that way, thats one data point
follow up nobody should copy my schedule, im describing it because people asked what a slow ramp looks like
monotherapy did work for me, just slower and smaller than i expected
i titrated over twice as many weeks as i did on semaglutide and that was right for me
amylin analogue, so its not a GLP-1 at all right
slightly off topic but dosing them on different days made no difference i could detect
i was sceptical about monotherapy and after 16 months im still sceptical, just less so
the numbers everyone quotes come from the combination, so comparing them to monotherapy logs is unfair
the slower titration in here is community habit as much as anything, the trials had their own schedule
how slow are people titrating this compared to retatrutide
lost 63lb over 25 months on the combination and i genuinely cannot apportion it
amylin not glp-1
did the nausea feel different to you or just less
two Janoshik results on the same HJ lot came back 97.4 and 98.1, closer than i expected